Quantity(Kilograms) | 1 - 10 | >10 |
Est. Time(days) | 5 | To be negotiated |
Clomiphene Citrate is a nonsteroidal fertility agent used to induce ovulation in infrequently ovulating or
anovulatory women,including patients with polycystic ovary syndrome (PCOS).The drug is effective at
producing ovulation in patients with an intact hypothalamic-pituitary-ovarian axis and with ovaries that
are capable of functioning normally. Clomiphene is often the first-line agent for these patients due to
the relative ease of use and low economic expense.Clomiphene is sometimes used as a diagnostic
tool to assess ovarian reserve. It is also administered to regulate the timing of ovulation in those
patients receiving donor insemination.Interestingly, clomiphene has been used to increase sperm
counts in male patients with idiopathic oligospermia.The FDA first approved clomiphene in 1967.
Clomid has a long half-life (possibly 5 days), so there is no need to split up doses throughout the day.
If has been used and Clomid is commenced 3 weeks after the last injection, I would estimate that
androgen levels are low enough to start sending the correct signals. If androgen levels are still a little
high, we need to start at a high enough amount thatwill work or help, even if androgen levels are still a
little high. Try 300mg on day 1;then use 100mg for the next 10 days;followed by 50mg for 10 days.
Product name | 2-Diethylaminoethyl hexanoate |
CAS NO | 10369-83-2 |
Purity | 99% |
Brand name | Natural Micron Pharm Tech |
Function
Clomid stimulates the hypothalamus to, in turn stimulant the anterior pituitary gland (aka hypophysis)
to release gonadotrophic hormones. The gonadotrophic hormones are follicle stimulating hormone
(FSH) and luteinizing hormone (LH - aka interstitial cell stimulating hormone (ICSH)). FSH stimulates
the testes to produce more test, and LH stimulates them to secrete more test. This feedback mechanism
is known as the hypothalamic-pituitary-testes axis (HPTA), and results in an increase of the body's own
production and blood levels rise, to, in part, compensate for the diminishing levels of exogenous steroids.
This is vital to minimise post cycle muscle losses.
Clomid does not, as is often thought, stimulate the release of natural , but rather works at reducing the
oestrogenic inhibition caused by the steroid cycle. It does this in a similar manner to the way it and
Nolvadex block oestrogen receptors in nipples to combat gyno development, i.e. by blocking the
oestrogen receptors in the hypothalamus and pituitary thus reducing the inhibition from the elevated
oestrogen. This allows LH levels to return to normal, or even above normal levels, and in turn,
natural levels to also normalise.